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ATCC primary human renal proximal tubule epithelial cells rptecs
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ATCC human renal proximal tubular epithelial cells
CHF protects against AI-AKI by modulating renal tubular <t>epithelial</t> cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).
Human Renal Proximal Tubular Epithelial Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ATCC human adrenocortical carcinoma cells
CHF protects against AI-AKI by modulating renal tubular <t>epithelial</t> cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).
Human Adrenocortical Carcinoma Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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rptec  (ATCC)
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ATCC rptec
CHF protects against AI-AKI by modulating renal tubular <t>epithelial</t> cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).
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ATCC pcs 400 010
CHF protects against AI-AKI by modulating renal tubular <t>epithelial</t> cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).
Pcs 400 010, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ATCC renal cortex proximal tubule epithelial cells
CHF protects against AI-AKI by modulating renal tubular <t>epithelial</t> cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).
Renal Cortex Proximal Tubule Epithelial Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ATCC normal human primary renal proximal tubule epithelial cells rptec
CHF protects against AI-AKI by modulating renal tubular <t>epithelial</t> cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).
Normal Human Primary Renal Proximal Tubule Epithelial Cells Rptec, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ATCC cvcl 0258 rptec atcc
CHF protects against AI-AKI by modulating renal tubular <t>epithelial</t> cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).
Cvcl 0258 Rptec Atcc, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ATCC pcs 400 010 huvec atcc
CHF protects against AI-AKI by modulating renal tubular <t>epithelial</t> cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).
Pcs 400 010 Huvec Atcc, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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CHF protects against AI-AKI by modulating renal tubular epithelial cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).

Journal: Frontiers in Molecular Biosciences

Article Title: Arsenic trioxide-induced acute kidney injury: OPA1- and Drp1-mediated mitochondrial dynamics imbalance, PINK1/Parkin-dependent mitophagy, and Chuanhuang Fang III

doi: 10.3389/fmolb.2026.1778855

Figure Lengend Snippet: CHF protects against AI-AKI by modulating renal tubular epithelial cell MQC in rats. (A, B) Mitochondrial membrane potential (Δψm) was detected by JC-1 staining. (C–F) OPA1 and Drp1 expression was assessed by immunofluorescence (scale bar = 40 µm) in rat kidney tissues. (G, H) Flow cytometry results of mitochondria ROS in rat kidney tissues. (I) Western blotting analysis of OPA1, Drp1, BNIP3, NIX, Parkin, PINK1, LC3B, Bcl-2 and BAX. (p <0.05, *p <0.01 vs CON or ATO or NAC, n=3).

Article Snippet: Human renal proximal tubular epithelial cells (HK-2, American Type Culture Collection, Manassas, VA, United States) were cultured in DMEM/F12 medium containing 10% fetal bovine serum and 1% penicillin-streptomycin at 37 °C in a humidified atmosphere with 5% CO 2 .

Techniques: Membrane, Staining, Expressing, Immunofluorescence, Flow Cytometry, Western Blot

Mechanism diagram of CHF against AI-AKI by regulating the OPA1- and Drp1-mediated mitochondrial dynamics imbalance and mitophagy in renal tubular epithelial cells.

Journal: Frontiers in Molecular Biosciences

Article Title: Arsenic trioxide-induced acute kidney injury: OPA1- and Drp1-mediated mitochondrial dynamics imbalance, PINK1/Parkin-dependent mitophagy, and Chuanhuang Fang III

doi: 10.3389/fmolb.2026.1778855

Figure Lengend Snippet: Mechanism diagram of CHF against AI-AKI by regulating the OPA1- and Drp1-mediated mitochondrial dynamics imbalance and mitophagy in renal tubular epithelial cells.

Article Snippet: Human renal proximal tubular epithelial cells (HK-2, American Type Culture Collection, Manassas, VA, United States) were cultured in DMEM/F12 medium containing 10% fetal bovine serum and 1% penicillin-streptomycin at 37 °C in a humidified atmosphere with 5% CO 2 .

Techniques: